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Hyperlipemia

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overview Blood fat mainly refers to the cholesterol and triglyceride in the serum. No matter the increase of the cholesterol or the increase of triglyceride or the increase of both of them, all called hyperlipemia. classification According to the outcome of the measurement of serum total cholesterol, triglyceride and high-density lipoprotein-cholesterol, hyperlipemia is divided into the following four types: 1. Hyoercholesteremia: The serum total cholesterol constant is increasing, and exceeds 5.2 mmol/L, but the constant of triglyceride is normal, which is to say the triglyceride is lower than 1.70mmol/L. 2. Hypertriglyceridemia: The content of serum triglyceride rises, and exceeds 1.70mmol/L, but the cholesterol content is normal, which means total cholesterol is lower than 5.2mmol/L. 3. Mixed type hyperlipemia: Both the contents of serum total cholesterol and triglyceride are increasing, which means the total cholesterol exceeds 5.2mmol/L and the triglyceride exceeds
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Urine protein

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Urine protein is the protein examined by the urine, which becomes muddy after the heat through acidize. 24 hours urine protein range is lower than or equal to 0.15g with normal people and by routine test, the urine protein is examined to be feminine. If the urine proteins are examined to be lower than 150ml/day, which means there is urine protein. It shows that the urine proteins are obviously increase in human bodies. It belongs to abnormal urine proteins. If the urine protein keeps positive, it often represents the lesion of urine proteins, thus clinic can decide the level of kidney damage and the effects of the kidney treatments according to the number of the positive of urine proteins. Therefore, the abnormal urine proteins occur. It must be controlled effectively and eliminated to prevent the worsen of the state of illness. If the glomerulus and kidney tubules are leted, such as the period of nephritis, kidney diseases and high blood pressure, which occult kidney arterioscler
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IgA Nephropathy Complications

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The course of IgA nephropathy varies from person to person. Some people have the disease for years with few problems. In fact, many cases may go undiagnosed. Other people develop one or more of the following complications: · End-stage uremia. If patients with IgA nephropathy don’t take a treatment timely, their renal function will be damaged gradually. Eventually, it will develop into severe uremia, which may lead to uremia high creatinine. · High blood pressure. Damage to your kidneys from IgA deposits can raise your blood pressure, and high blood pressure can cause further damage to your kidneys. · High cholesterol. High levels of cholesterol may increase your risk of a heart attack. · Acute kidney failure. If your kidneys lose their filtering ability due to IgA deposits, waste products build up quickly in your blood. · Chronic kidney failure. IgA nephropathy can cause your kidneys to gradually stop functioning. In such cases, permanent dialysis or a kidney transplant is
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Prevention of renal calcification

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Because the blood calcium rises, calcium salts deposited in the renal parenchyma, which causes kidney functions obstacle. When calcification, it can show the motting calcification or the renal calcification shadows in the urinary tract plain film. Kidney calcification manifests to be early kidney calices margin to be the shape of rats biting in the urinary tract radiography. While, how to prevent calcification? (1) Preventing overwork, spirit high pressure: Overwork, work late into the night, the pressure of mantang is big, all of them can make the chronic nephritis aggravated. We should cultivate good life styles, keep regular lives. At ordinary times, we should possible arrange life and work and rest regime, attending more appropriate activities, reinforce physical excercise, but we should prevent overwork. Rational nutrition, strengthen constitution and the immunity of human bodies. Pay attention to personnel hygiene and the clean of environment health, cultivate good life
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Treatments of Nephrapostasis

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We all know something about nephrapostasis. The following are the treatments of nephrapostasis. 1. Treatments of the whole body Patients should stay in bed, transfuse and drink more water, keep daily volume of urine to be above 1.5L, which is good for the ejectment of inflammation products. Patients should also pay much attention to daily diet, they should eat foods that are easy to digestion and full of quantity of heat and vitamins. 2. Treatments of antibacterial agents Medicines that can be choosed are: ①SMZ-TMP is effective to the positive or negative bacterium of Gelan except for pseudomonas aeruginosa. ② Quinolones antibacterial spectrum is widely used, its effect is strong, virulence is little, the clinic has already been used widely, apart from using to children and pregnant women. ③ Penicillin ④ The first and second cephalosporin can be used to produce enzyme staphy lococcus infection. The second and third cephalosporin do good outstandingly to severe gram n
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Urine Occult Blood

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Urine occult blood is examined through routine urianlysis, there is a red blood cell in the urine occult blood. If the red blood cells increase, (normal is negative), and the number of it is few, it can be called urine occult blood. While urine occult blood itself can be an index to decide whether it is negative or positive and if it is positive, how many + are there in it. We should consider the problems of the kidneys or it may be caused by urinary tract infection if the urine occult blood occurred generally. Generally speaking, urine occult blood are caused by the following three reasons: Inflammation, stones and tumour. For the area of inflammation, such as glomerulonephritis, pyelonephritis, urocystitis and so on. In the examination of urine, it may be have hematuresis, and also occult blood. The stones themselves, no matter the kidneys, ureter, or cystolith all can cause occult blood. Other conditions, such as physiological and constipation may also cause occult blood.
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Complications of IgA Nephropathy

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IgA nephropathy can occur at any age, even in childhood. More men are affected than women. Although found all over the world, IgA nephropathy is more common among Caucasians and Asians. It is one of the most common diseases of the kidney, other than those caused by diabetes or high blood pressure. There are many complications of IgA nephropathy. High blood pressure Damage to your kidneys from IgA deposits can raise your blood pressure, and high blood pressure can cause further damage to the kidneys. They influence each other. Acute kidney failure If your kidneys lose their filtering ability due to IgA deposits, the waste products and toxins accumulate quickly in the blood. When this happens, you may need to undergo temporary dialysis. Chronic kidney failure If IgA Nephropathy can not be controlled well, the illness condition will keep worsening. The kidneys will gradually stop functioning. More and more waste products and toxins build up in the blood, a series of symptom
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Prolonged Sitting Increases Risk of Kidney Disease

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Individuals who sit for several hours of the day not only put themselves at risk for obesity and sore limbs, but also increase the risk of developing diabetes and chronic kidney disease (CKD). CKD is a condition where the kidneys aren't able to properly filter blood, which can lead to waste build-up within the body and eventually kidney failure. Known causes of chronic kidney disease include high blood pressure and diabetes. Prolonged sitting will cause obesity, thus causing diabetes and CKD. Women are able to lower their risk of developing chronic kidney disease by sitting fewer hours a day -- their risk fall by more than 30 percent when they cut sitting time from eight hours to three hours daily. Men also see a benefit in slashing sitting time from eight to three hours, though their risk decreased less by just 15 percent. However, men are more likely to be able to offset the increased risk of chronic kidney disease by exercising, compared with women. Prolonged sitting is
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Precautions for PKD in Daily Life

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PKD is a hereditary kidney disease. To PKD patients, precautions in daily life are more important than the short time treatment in clinic. The enlarge of the cysts is caused by many factors, and patients should pay more attention to the factors so as to control the lesions. 1. Prevent cold PKD needs the extra care and family’s considerate nurse. But these can’t stop the enlargement of cysts. If PKD patients get cold, especially the repeated cold, they will suffer severe kidney damage. Cold will aggravate the damaged kidney. It is necessary to prevent cold in case of the aggravation. 2. Prevent an injury The gradual growth of cysts will cause the increase of the inner pressure of cysts, causing the double kidneys getting larger and intra-abdominal pressure increasing. If there is any light injury, such as sprain, bumping, injury from fall, which will increase the inner pressure of the abdominal cavity. And the injury will also directly impact the cysts in kidneys, causing the
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Polycystic Kidney Disease and Polycystic Liver Disease

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Polycystic kidney always appear with polycystic liver. Polycystic liver refers to the liver with more than three cysts, distributing along the hepatic duct. If you have polycystic kidney, you probably have polycystic liver. Polycystic liver has the family hereditary tendency. Female is more than male. The morbidity of 3 years old is about 20% and 70 years old, 75%. If patients with successfully allograft kidney transplantation live for more than ten years, they will suffer from polycystic liver. The hyperplasia of the epithelium and mesenchyme in bile duct causes the multiple cysts in the liver tissues, namely polycystic liver disease. The bile duct dilation, congenital hepatic fibrosis and female hormone have a certain effect on PLD. Cysts of PKD are of different size. Smaller can be found by micro-scope, bigger could occupy in the whole abdominal cavity. And liver will become huge from normal size. Abdominal B-ultrasound and CT scan are the reliable method to examine whether
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