Health and Exercise

overview Blood fat mainly refers to the cholesterol and triglyceride in the serum. No matter the increase of the cholesterol or the increase of triglyceride or the increase of both of them, all called hyperlipemia. classification According to the outcome of the measurement of serum total cholesterol, triglyceride and high-density lipoprotein-cholesterol, hyperlipemia is divided into the following four types: 1. Hyoercholesteremia: The serum total cholesterol constant is increasing, and exceeds 5.2 mmol/L, but the constant of triglyceride is normal, which is to say the triglyceride is lower than 1.70mmol/L. 2. Hypertriglyceridemia: The content of serum triglyceride rises, and exceeds 1.70mmol/L, but the cholesterol content is normal, which means total cholesterol is lower than 5.2mmol/L. 3. Mixed type hyperlipemia: Both the contents of serum total cholesterol and triglyceride are increasing, which means the total cholesterol exceeds 5.2mmol/L and the triglyceride exceeds

Urine protein is the protein examined by the urine, which becomes muddy after the heat through acidize. 24 hours urine protein range is lower than or equal to 0.15g with normal people and by routine test, the urine protein is examined to be feminine. If the urine proteins are examined to be lower than 150ml/day, which means there is urine protein. It shows that the urine proteins are obviously increase in human bodies. It belongs to abnormal urine proteins. If the urine protein keeps positive, it often represents the lesion of urine proteins, thus clinic can decide the level of kidney damage and the effects of the kidney treatments according to the number of the positive of urine proteins. Therefore, the abnormal urine proteins occur. It must be controlled effectively and eliminated to prevent the worsen of the state of illness. If the glomerulus and kidney tubules are leted, such as the period of nephritis, kidney diseases and high blood pressure, which occult kidney arterioscler

The course of IgA nephropathy varies from person to person. Some people have the disease for years with few problems. In fact, many cases may go undiagnosed. Other people develop one or more of the following complications: · End-stage uremia. If patients with IgA nephropathy don’t take a treatment timely, their renal function will be damaged gradually. Eventually, it will develop into severe uremia, which may lead to uremia high creatinine. · High blood pressure. Damage to your kidneys from IgA deposits can raise your blood pressure, and high blood pressure can cause further damage to your kidneys. · High cholesterol. High levels of cholesterol may increase your risk of a heart attack. · Acute kidney failure. If your kidneys lose their filtering ability due to IgA deposits, waste products build up quickly in your blood. · Chronic kidney failure. IgA nephropathy can cause your kidneys to gradually stop functioning. In such cases, permanent dialysis or a kidney transplant is

Because the blood calcium rises, calcium salts deposited in the renal parenchyma, which causes kidney functions obstacle. When calcification, it can show the motting calcification or the renal calcification shadows in the urinary tract plain film. Kidney calcification manifests to be early kidney calices margin to be the shape of rats biting in the urinary tract radiography. While, how to prevent calcification? (1) Preventing overwork, spirit high pressure: Overwork, work late into the night, the pressure of mantang is big, all of them can make the chronic nephritis aggravated. We should cultivate good life styles, keep regular lives. At ordinary times, we should possible arrange life and work and rest regime, attending more appropriate activities, reinforce physical excercise, but we should prevent overwork. Rational nutrition, strengthen constitution and the immunity of human bodies. Pay attention to personnel hygiene and the clean of environment health, cultivate good life

We all know something about nephrapostasis. The following are the treatments of nephrapostasis. 1. Treatments of the whole body Patients should stay in bed, transfuse and drink more water, keep daily volume of urine to be above 1.5L, which is good for the ejectment of inflammation products. Patients should also pay much attention to daily diet, they should eat foods that are easy to digestion and full of quantity of heat and vitamins. 2. Treatments of antibacterial agents Medicines that can be choosed are: ①SMZ-TMP is effective to the positive or negative bacterium of Gelan except for pseudomonas aeruginosa. ② Quinolones antibacterial spectrum is widely used, its effect is strong, virulence is little, the clinic has already been used widely, apart from using to children and pregnant women. ③ Penicillin ④ The first and second cephalosporin can be used to produce enzyme staphy lococcus infection. The second and third cephalosporin do good outstandingly to severe gram n

Urine occult blood is examined through routine urianlysis, there is a red blood cell in the urine occult blood. If the red blood cells increase, (normal is negative), and the number of it is few, it can be called urine occult blood. While urine occult blood itself can be an index to decide whether it is negative or positive and if it is positive, how many + are there in it. We should consider the problems of the kidneys or it may be caused by urinary tract infection if the urine occult blood occurred generally. Generally speaking, urine occult blood are caused by the following three reasons: Inflammation, stones and tumour. For the area of inflammation, such as glomerulonephritis, pyelonephritis, urocystitis and so on. In the examination of urine, it may be have hematuresis, and also occult blood. The stones themselves, no matter the kidneys, ureter, or cystolith all can cause occult blood. Other conditions, such as physiological and constipation may also cause occult blood.

IgA nephropathy can occur at any age, even in childhood. More men are affected than women. Although found all over the world, IgA nephropathy is more common among Caucasians and Asians. It is one of the most common diseases of the kidney, other than those caused by diabetes or high blood pressure. There are many complications of IgA nephropathy. High blood pressure Damage to your kidneys from IgA deposits can raise your blood pressure, and high blood pressure can cause further damage to the kidneys. They influence each other. Acute kidney failure If your kidneys lose their filtering ability due to IgA deposits, the waste products and toxins accumulate quickly in the blood. When this happens, you may need to undergo temporary dialysis. Chronic kidney failure If IgA Nephropathy can not be controlled well, the illness condition will keep worsening. The kidneys will gradually stop functioning. More and more waste products and toxins build up in the blood, a series of symptom

Individuals who sit for several hours of the day not only put themselves at risk for obesity and sore limbs, but also increase the risk of developing diabetes and chronic kidney disease (CKD). CKD is a condition where the kidneys aren't able to properly filter blood, which can lead to waste build-up within the body and eventually kidney failure. Known causes of chronic kidney disease include high blood pressure and diabetes. Prolonged sitting will cause obesity, thus causing diabetes and CKD. Women are able to lower their risk of developing chronic kidney disease by sitting fewer hours a day -- their risk fall by more than 30 percent when they cut sitting time from eight hours to three hours daily. Men also see a benefit in slashing sitting time from eight to three hours, though their risk decreased less by just 15 percent. However, men are more likely to be able to offset the increased risk of chronic kidney disease by exercising, compared with women. Prolonged sitting is

PKD is a hereditary kidney disease. To PKD patients, precautions in daily life are more important than the short time treatment in clinic. The enlarge of the cysts is caused by many factors, and patients should pay more attention to the factors so as to control the lesions. 1. Prevent cold PKD needs the extra care and family’s considerate nurse. But these can’t stop the enlargement of cysts. If PKD patients get cold, especially the repeated cold, they will suffer severe kidney damage. Cold will aggravate the damaged kidney. It is necessary to prevent cold in case of the aggravation. 2. Prevent an injury The gradual growth of cysts will cause the increase of the inner pressure of cysts, causing the double kidneys getting larger and intra-abdominal pressure increasing. If there is any light injury, such as sprain, bumping, injury from fall, which will increase the inner pressure of the abdominal cavity. And the injury will also directly impact the cysts in kidneys, causing the

Polycystic kidney always appear with polycystic liver. Polycystic liver refers to the liver with more than three cysts, distributing along the hepatic duct. If you have polycystic kidney, you probably have polycystic liver. Polycystic liver has the family hereditary tendency. Female is more than male. The morbidity of 3 years old is about 20% and 70 years old, 75%. If patients with successfully allograft kidney transplantation live for more than ten years, they will suffer from polycystic liver. The hyperplasia of the epithelium and mesenchyme in bile duct causes the multiple cysts in the liver tissues, namely polycystic liver disease. The bile duct dilation, congenital hepatic fibrosis and female hormone have a certain effect on PLD. Cysts of PKD are of different size. Smaller can be found by micro-scope, bigger could occupy in the whole abdominal cavity. And liver will become huge from normal size. Abdominal B-ultrasound and CT scan are the reliable method to examine whether

For every patient with kidney disease to treat, we will ask them a question: What disease do you have? Do you think this is kidding? of course not! Anyway, all kidney disease? It is no exaggeration to say that many patients with kidney disease do not know what disease they have. There was once a patient with nephropathy, a typical manifestation of nephrotic syndrome, and it was very swollen when admitted to hospital. Urine protein 4+ and plasma albumin remained only 11.2 g/L. He has been using hormone therapy, but the effect has not been good, the condition is getting heavier, he did not know why: "In the last hospital, my bed next door is also kidney synthesis, all aspects are similar to me, how he uses hormones like So, I'm not effective with hormones?" We did a series of tests for him (his constitution is not suitable for renal puncture), especially positive for the anti-phospholipase A2 antibody receptor, so that we eventually diagnosed as membranous neph

x According to the Global Disease Burden Report, the important cause of chronic kidney disease-related life loss has risen from 25 in 1990 to 17 in 2015, and the disability rate of kidney disease has also grown at an annual rate of 1.3%. It has brought great burden and influence to patients, families and society. Kidney disease has become a major global health issue. With the current medical level, there is still a long way to go to improve the prognosis of the global kidney disease. At the same time, letting the treatment of kidney disease worse, the patient itself has also added a fire to the disease, so that the condition is even more uncontrolled, kidney disease has four fears, to see if you have done it all. A fear: you ignore the symptoms of the disease The condition of kidney disease is relatively hidden and the symptoms are not obvious. Unless you look closely. The main performance is reflected in urine. The color of the urine, if it is bloody, soy sauce, or flesh-c

Recently, a patient with chronic renal failure, with a creatinine concentration of 172 μmol/L, entered the third phase of kidney disease. It is regrettable to read his previous inspection report. At the 2016 medical exam, creatinine was already 109, and creatinine was 115 at 8 months before. Surprisingly, almost two years later, he had not taken any medicine or had any treatment. I asked him: "In September of the previous year, your creatinine exceeded, and you should know?" "know." "How hasn't been treated?" “I think that the high creatinine point is okay, then I'm going to have a bit high transaminase and my blood pressure is always high. Our normal creatinine value is 54-106, and the feeling of a hundred nodded creatinine is not big, but now creatinine is getting higher and higher. It's up." "At that time, you should consult the Department of Nephrology." "I was seeing a doctor at the time. I had a buddy, a docto

There are often patients in the background to leave me a message: Doctor, I am currently creatinine XXX, how many times have been dialysis, I still have the possibility of recovery? Such a problem, if answered from a professional clinical point of view, the result is often a probable event: a few percent of patients can get better, and a few percent of patients show specific phenomena... various uncertainties The result will follow. The implication is that even if the two people are close together, the outcome may be very different. How to judge whether there is treatment value in the condition? As long as the renal ECT shows glomerular filtration rate greater than 10, it shows that there is therapeutic value. If the patient has progressed to uremia, the kidney function is expected to return to normal levels, which is basically impossible to do. After all, the disease itself cannot be compared with those with creatinine of 200 or more. The purpose of treatment at this time is

IgA nephropathy indicates that in the glomerular mesangial area, it is mainly based on IgA or IgA deposition. It is the primary immunotactoid glomerulopathy with or without other immune globulins depositing in the glomerular mesangial area. It clinically manifests the repeated attack of gross hematuria and microscopic hematuria. It can be accompanied by the proteinuria in varying degrees. Some patients have the high blood pressure or renal insufficiency. While what is the reasonable diet of IGA nephritis? 1. The provide of protein The diet of IgA nephropathy should be confirm the intake of protein based on the degree of renal function damage. The patients, who have long term course of disease and the damage of the kidney is not serious, can eat the protein without close restraint. But they should not eat more than 1g per kilogram weight daily. Good protein should above 50%. 2. The provision of energy Some patients are limited to eat less protein, their provision of energy

What is the diet to improve GFR? Increasing GFR effectively means a lot for kidney disease patients and this may indicate they can live far away from dialysis. Diet is an important part for kidney disease patients to improve GFR effectively, but before learning the die to improve GFR, let’s found out what GFR is and how kidney disease cause low GFR. What is GFR? GFR is the abbreviation of Glomerular Filtration Rate which is a test used to check how well the kidneys are working. Normally, GFR ranges from 90-120mL/min/1.73m2. And for kidney disease patients, they usually have low GFR. the diet to improve GFR Soy Protein · Protein is an important part of any diet. It builds muscle and repairs damage in the body. Unfortunately, many sources of protein are harmful to someone at risk for kidney disease. When a diabetic, for example, eats animal protein, the levels of glomerular filtration rate (GFR) shoot up and reduce his function. After consuming soy protein, however, the person's GF

Nephritis is the inflammatory lesion of non suppurative of the both sides of the kidneys. There will be edema, high blood pressure, and other symptoms because of the renal corpuscle damage, which is the most commen kind of kidney diseases. There are a great variety of nephritis, such as acute glomerulus nephritis, chronic glomerulus nephritis, nephropyelitis, latent glomerulonephritis, henoch-schonlein purpura nephritis (anaphylactic purpura nephritis), lupus erythematosus nephritis (lupus nephritis). Symptoms of nephritis premonitory symptom Most of the patients have pioneer history of infection one month of premorbid, the onset of the illness is mainly all of a sudden, but it can also be recessively slow onset. urine abnormal It is usually begain with oliguria, or oliguria little by little, even anuria. It can be accompanied by gross hematuria meanwhile, the time of duration is unequal. But the microscopic hematuria sustains. The change of routine urine test and acute gl

The main cause of the kidney stones is diet. It is caused by too much intake of the related elements, which causes the stones in the diet. While what tests should be done to the kidney stones? 1. X-ray examination: X-rat examination is an important method to diagnose the kidneys and the ureteral calculus. About more than 95% lithangiuria can be developed to be the X-ray plain. Supplemented by the excretory or retrograde pelvis ureter radiography, it can be seen wether there are obstruction or the degree of the obstruction in the part of the stone. It can confirm the therapeutic schedule by ensure whether it does good to the split renal function, distinguishing the calcification shadow coming from the outside urinary tract and eliminating other lesions of the upper urinary tract. After the treatment, the size of the stones part, comparison of the number and so on, all have important values. 2. Laboratory examination: There are red blood cells, hemameba or crytalluria pH in the ur

The Committee on IgA nephropathy in Japan has published new clinical guidelines (2nd edition) for the diagnosis and treatment of patients with this disease. The nonspecific therapeutic approach involves a reduction of dietary intake of protein in patients with IgA nephropathy who have developed renal failure. At present, the most important therapeutic goal in patients with IgA nephropathy is the control of hypertension. It has been assumed that removal of tonsillar tissues might reduce the production of polymeric IgA and decrease the frequency of renal parenchymal damage resulting from episodes of macroscopic hematuria and proteinuria. Although there have been no randomized controlled trials (RCT) of tonsillectomy, these are necessary to determine the efficacy of tonsillectomy in patients with IgA nephropathy. The treatments of IgA nephropathy are as follows: 1. The hypertensives should control blood pressure and maintain within usual level to remiss hemodynamics and aggravate t

Hypertensive nephropathy is kidney dysfunction caused by persistent high blood pressure. This is a common form of renal disease and is a frequent cause of damage so severe that the patient requires renal dialysis to take over for failing kidneys. There are some measures available to prevent hypertensive nephropathy, or to address the damage early to improve quality of life for the patient. Some introductions in detail are as follows: Severe edema Patients with Hypertensive Nephropathy usually have edema which may distribute in the whole body. Severe one may also accompany with oliguria, high blood pressure and mild azotemia. Hence, we know edema especially the severe edema is another obvious complications besides high blood pressure. Urine change Proteinuria and hematuria appear along with the increase of nocturna. Hypertensive Nephropathy is easy to develop into renal failure gradually if his proteinuria and hematuria can’t be controlled in the early stage. Urine change als

Acute glomerulonephritis is called acute nephritis for short. Some people call it acute nephritic syndrome because of the difference of pathogenesis. It is a diffuse glomerular non suppurative inflammatory lesion caused by the immunoreaction after the infections. It is mainly edema, oliguria, hematuresis and high blood pressure clinically. The sick children usually have influenza, amygdalitis or purulent skin and other precursor diseases in the premorbid period. This disease is the most common kidney disease in the infantile period, which is commonly seen with 3 to 8 years old children, children under 2 years old are rarely seen. The prognosis is usually well. The course of disease is six months or one year, patients who can develope to the chronic nephritis are rarely. While what symptoms will occur with acute glomerulonephritia? 1. The majority of the patients have resiratory tract infection, skin infection, some virus infections and other precursor history of infections. 2.